Authors describe the in vivo behavior of mitochondria at the growth cone of elongating retinal axons in zebrafish.

Authors show interneuron defects in a new mouse model of premature birth that combines prenatal inflammatory and postnatal hypoxic insults.

Authors identify capsaicin receptor TRPV1 as a key regulator of Ca2+ entry into axoplasm that is required for developmental degeneration modeled by NGF withdrawal from sensory neurons of the dorsal root ganglion in vitro.