Editor's Pick
Results of this study suggest that decreased responding to food-associated cues when food becomes less desirable are due to changes in the excitability of neurons in the nucleus accumbens.
Editor's Pick: Does Learning a Second Language Rely on Familiarity with Native Language Orthography?
Authors tested whether native English-speaking adults could learn a second, visually atypical writing system, HouseFont, and used neuroimaging data to assess the location of any learning effects.
Authors provide the first functional description of the emergence of neocortical fast ripples in hippocampal epilepsy and show that cross-frequency coupling might be a fundamental mechanism underlying the spreading of epileptic activity.
Authors show that excitation and inhibition of pyramidal neurons in different basolateral amygdala subdivisions promote auditory fear memory formation.
Authors used a novel method to show that selectively activating cholecystokinin-expressing GABAergic neurons subtly affects emotional behavior but surprisingly enhances multiple memory and cognitive processes.
Authors identify capsaicin receptor TRPV1 as a key regulator of Ca2+ entry into axoplasm that is required for developmental degeneration modeled by NGF withdrawal from sensory neurons of the dorsal root ganglion in vitro.
Authors describe an unexpected effect of in utero electroporation of plasmid DNA on the distribution of microglia within the developing forebrain.
Authors addressed the question 'what, if anything, is rodent prefrontal cortex?' by carrying out a survey of prefrontal cortex researchers and uncovered points of agreement as well as differences in regard to what brain areas constitute "prefrontal cortex" in rodents."
Authors show that axons of inhibitory neurons have distinctive structural and molecular features that contrast with those of the majority of myelinated excitatory axons in human neocortex.
Authors functionally defined immediate-early astrocytes (ieAstrocytes) as an in vivo astrocyte population, modulated by S1P signaling, in a mouse model of multiple sclerosis.
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