Authors show that key features of the microglial activation pattern observed after status epilepticus may not be related to the epileptogenic process.
Authors show interneuron defects in a new mouse model of premature birth that combines prenatal inflammatory and postnatal hypoxic insults.
Authors show that although handedness impacts the accuracy of hand movement control, it has virtually no influence on the ability to predict the visual consequences of hand movements.
Dr. Emanuela De Falco tells the story about her co-first author eNeuro paper that describes distinct neural activity states that occur in the medial prefrontal cortex while rats perform an odor span task of working memory capacity.
Authors show that one olfactory cortical structure, the olfactory tubercle, is unique among olfactory cortices in that it lacks feedback projections to the olfactory bulb.
See the most shared articles of May/June 2019; Volume 6,Issue 3
Authors show that blocking endogenous Bone Morphogenetic Protein 4 signaling via a selective pharmacological approach promotes oligodendroglial differentiation and increases the rate of remyelination after a central demyelinating insult in vivo.
Authors tested whether native English-speaking adults could learn a second, visually atypical writing system, HouseFont, and used neuroimaging data to assess the location of any learning effects.
Authors provide the first functional description of the emergence of neocortical fast ripples in hippocampal epilepsy and show that cross-frequency coupling might be a fundamental mechanism underlying the spreading of epileptic activity.
PhD Candidate Ruilong Hu and Dr. Ricardo Araneda tell the story about their eNeuro publication that showed that the two predominant subtypes of inhibitory neurons in the main olfactory bulb, the granule cells and periglomerular cells, exhibit subthreshold resonance mediated by hyperpolarization-activated currents (Ih), as opposed to the primary output neurons, the mitral cells.
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- Early Biomarker for Motor Skill Consolidation Defects in Huntington’s Disease Mouse Model
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